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972-8. M. C. (2002). Towards a global historical emission inventory for selected PCB congeners--a mass balance approach. 1. Global production and consumption. The Science of the Total Environment, 290, 1-3, (May 2002), pp. 181-98. ; Bergman, A. J. (1998). Interactions of persistent environmental organohalogens with the thyroid hormone system: mechanisms and possible consequences for animal and human health. Toxicology and Industrial Health, 14, 1-2, (January-April 1998), pp. 59-84. Brucker-Davis, F.

871-6. ; Wilhelm, M. & Twardella, D. (2009). Perfluorinated compounds—exposure assessment for the general population in Western countries. International Journal of Hygiene and Environmental Health, 212, 3, (May 2009), pp. 239– 270. R. A. (2004). Changes in some hormones by low doses of di (2-ethyl hexyl) phthalate (DEHP), a commonly used plasticizer in PVC blood storage bags and medical tubing. Indian Journal of Medical Research 119, 4, (April 2004), pp. 139–144. ; Bansal, R. T. (2004). Polychlorinated biphenyls (PCBs) exert thyroid hormone-like effects in the fetal rat brain but do not bind to thyroid hormone receptors.

2005). Effects of perchlorate exposure on resting metabolism, peak metabolism, and thyroid function in the prairie vole (Microtus ochrogaster). Environmental Toxicology and Chemistry, 24, 3, (May 2005), pp. 678-84. ; Kuroda, Y. & Koibuchi, N. (2002). Polychlorinated biphenyls suppress thyroid hormone-induced transactivation. Biochemical and Biophysical Research Communications, 299, 3, (December 2002), pp. 384-8. ; Blaha, L. & Holoubek, I. (2006). Environmental xenobiotics and nuclear receptors--interactions, effects and in vitro assessment.

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